programmed puller p-97 (Sutter Instrument Company)
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Sutter Instrument Company
programmed puller p-97
Programmed Puller P 97, supplied by Sutter Instrument Company, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/programmed+puller+p-97/micropipette+puller+p+97/pm40180159-49-20-22
Average 90 stars, based on 1 article reviews
Programmed Puller P 97, supplied by Sutter Instrument Company, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/programmed+puller+p-97/micropipette+puller+p+97/pm40180159-49-20-22
Average 90 stars, based on 1 article reviews
programmed puller p-97 - by Bioz Stars,
2026-10
90/100 stars
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other:Article Title: Spinal cord trunk preparation for analyzing cross-segmental primary afferent signal transmission and modulation. Article Snippet: Background: The spinal cord dorsal horn is pivotal for primary afferent signal transmission and modulation.. Primary afferent fibers from each dorsal root arrive at the dorsal horn and travel 1–2 segments caudally and rostrally.. Usually, in vitro spinal cord slices or in vivo preparations are employed for primary afferent stimulation and patch-clamp recordings to assess input signals. Article Title: Presynaptic GABA B receptors differentially modulate GABA release from cholecystokinin and parvalbumin interneurons onto CA1 pyramidal neurons: A cell type-specific labeling and activating study. Article Snippet: Combining cell type-specific optogenetics and whole cell recordings on mouse acute hippocampal slices, we compared GABA release from cholecystokinin-expressing (CCK) and parvalbumin-expressing (PV) interneurons onto CA1 pyramidal neurons.. Baclofen, a selective GABAB receptor agonist, inhibited GABAergic synaptic transmission greater from CCK terminals, compared to that from PV terminals.. The N-type calcium channels on CCK and P/Q-type calcium channels on PV terminals contributed to the GABAB receptor-mediated inhibition, respectively. |